Turkish Journal of Geriatrics 2011 , Vol 14 , Issue 4
LETTER TO THE EDITOR
Volkan GENÇ, Elvan Onur KIRIMKER, Şiyar ERSÖZ, Ahmet Serdar KARACA, Erkin OROZAKUNOV, Gökhan ÇİPE
Ankara University Faculty of Medicine Department of General Surgery ANKARA

Introduction

TO THE EDITOR

Uterine prolapse is the herniation of uterus into or through the vagina. In UK, the annual incidence of hospital admission with prolapse is 20.4/10000. Confirmed risk factors are older age higher parity, vaginal delivery, race, family history, obesity and constipation [1]. Menopause, long second stage of labor, musculoskeletal disease trauma, smoking and increased intra-abdominal pressure are possible risk factors [2],[3]. Common complaints and symptoms are feeling of a vaginal bulge and pressure urinary or fecal incontinence, feeling of incomplete voiding or defecation, weak or prolonged urinary stream, dyspareunia and lack of sexual sensation [4],[5].

A 74-year-old woman who was treated due to esophageal variceal bleeding in gastroenterology service was referred to our surgical department for her recurrent umbilical hernia. She was diagnosed with chronic liver disease fifteen years ago and had six vaginal deliveries. On physical examination, recurrent umbilical hernia massive ascites and uterovaginal prolapse (Figure 1) were detected. Prolapse developed gradually during the last five years whereas formation of ascites had started 10 years ago. Operation for recurrent umbilical hernia and uterovaginal prolapse was proposed but she declined.

Multiparity causes recurrent levator ani injuries and high intraabdominal pressure certainly facilitates uterine prolapsed [6]. Nevertheless ascites has the main role in intraabdominal pressure increase. Umbilical hernia secondary to massive ascites is a common pathology with a high recurrence rate [7]. We believe that this is the first case in English literature that demonstrates the association between chronic massive ascites and uterovaginal prolapse.


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Figure 1: Appearance of massive uterovaginal prolapse.

Reference

1) Mant J, Painter R, Vessey M. Epidemiology of genital prolapse: observations from the Oxford family planning association study. Br J Obstet Gynaecol 1997;104:579-85. (PMID: 9166201).

2) Swift SE, Woodman P, O’Boyle A, et al. Pelvic organ support study (POSST): the distribution, clinical definition and epidemiology of pelvic organ support defects. Am J Obstet Gynecol 2005;192:795-806. (PMID: 15746674).

3) Olsen A, Smith VJ, Bergstrom JO, Colling JC, Clark AL. Epidemiology of surgically managed pelvic organ prolapse and urinary incontinence. Obstet Gynecol 1997;89:501-6. (PMID: 9083302).

4) Doshani A, Teo REC, Mayne CJ, Tincello DG. Uterine prolapse. BMJ 2007;335:819-23. (PMID: 17947787).

5) Schaffer JI, Wai CY, Boreham MK. Etiology of pelvic organ prolapse. Clin Obstet Gynecol 2005;48:639-47. (PMID: 16012231).

6) Davis JD, Carroccio S. Massive uterovaginal prolapse in a young nulligravida with ascites: a case report. J Reprod Med 2007;52:727-9. (PMID: 17879835).

7) McKay A, Dixon E, Bathe O, Sutherland F.Umbilical hernia repair in the presence of cirrhosis and ascites: results of a survey and review of the literature. Hernia 2009;13:461-8. (PMID: 19652907).